Portal:Disease/COVIDPathways
From WikiPathways
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|width=100px|{{#pwImage:Pathway:WP4912|250px||SARS coronavirus and innate immunity}} | |width=100px|{{#pwImage:Pathway:WP4912|250px||SARS coronavirus and innate immunity}} | ||
|width=100px|{{#pwImage:Pathway:WP4927|250px||COVID-19, thrombosis and anticoagulation}} | |width=100px|{{#pwImage:Pathway:WP4927|250px||COVID-19, thrombosis and anticoagulation}} | ||
+ | |width=100px|{{#pwImage:Pathway:WP4883|250px||SARS-CoV-2 and Angiotensin-converting enzyme 2 (ACE2) receptor: Molecular mechanisms and potential therapeutic target}} | ||
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Revision as of 10:30, 18 May 2020
This special subset of disease pathways is being highlighted during the current COVID-19 crisis. This content is released under a CC0 waiver to be freely used, reused and distributed. Let us know if you add a new pathway or want to recommend one for this collection. |
Image does not exist Collaborative project for curation biological processes involved in the COVID-19 disease after SARS-Cov-2 infection. |
Image does not exist Lipid metabolism alternations that are related to infection by the corona virus. |
Image does not exist SARS-CoV-2 includes a novel Orf10 that interacts with muliple members of the Cullin 2 ubiquitin ligase complex. |
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Image does not exist The induction and signaling of type I interferon with specific SARS-CoV-2 protein and relevant drug. |
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Image does not exist Orf3a from SARS-CoV has been shown to bind TRAF3 and activate the NLRP3 inflammasome. |
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Image does not exist Experimental pathway to investigate overlap of virus-host interaction and adverse outcome pathways |
Image does not exist Virus-host interactome and proteomic survey of PMBCs from COVID-19 patients reveal potential virulence factors influencing SARS-CoV-2 pathogenesis |
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More about ACE2
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