Oxidative stress in pulmonary tissue (Homo sapiens)

From WikiPathways

Jump to: navigation, search
ArcPathVisio Brace Ellipse EndoplasmicReticulum GolgiApparatus HexagonPathVisio MimDegradation Mitochondria Octagon PentagonPathVisio Rectangle RoundedRectangle SarcoplasmicReticulum TriangleEquilateralEast TrianglePathVisio none Cigarette smokeParticulate matterEmission from biomassfuel and combusitonNitrogen oxidesPollen grainsOzoneIndustrial smokeSulphur oxidesCarbon monoxidePesticidesStressorsApoptosisKE3: Cell death and cycle arrestKE2: Inflammation and pro-fibrotic stateAO1: Idophatic pulmonary fibrosisKE1: Increased oxidative stressKE6: ER stressKE4: Mitrochondrial dysfucntionRelease of pro-inflammatory cytokinesAO2: COPDAO3: Acute respiratory distresssyndromeKE5: Collagen production and reduced ECMAO4: AsthmaProfibrotic pathwayMIE1: ROS production upregulationSenescenceROS damage pathwayChemokine signalling pathwayName: Oxidative stress in pulmonary tissueOrganism: Homo sapiens


Description

Adverse Outcome Pathway (AOP) for stressor induced increase in ROS production in pulmonary tissue.

Try the New WikiPathways

View approved pathways at the new wikipathways.org.

Quality Tags

Ontology Terms

No Tags!
 

Bibliography

  1. Sajal Chakraborti, Narasimham L. Parinandi, Rita Ghosh, Nirmal K. Gangul, Tapati Chakraborti; ''Oxidative Stress Mechanisms in the Pathogenesis of Environmental Lung Diseases''; Pubmed, 2019 PubMed Europe PMC Scholia

History

View all...
CompareRevisionActionTimeUserComment
136622view11:28, 18 February 2025EweitzClear dimension cache
136621view11:27, 18 February 2025EweitzEconomize layout
135313view21:29, 16 August 2024KhanspersOntology Term : 'oxidative stress response pathway' added !
133233view07:50, 10 June 2024BurritacoEndogenous ROS production was deleted and lipid membrane oxidation was removed.
131847view15:11, 4 June 2024BurritacoNew pathway

External references

DataNodes

View all...
Name  ↓Type  ↓Database reference  ↓Comment  ↓
AO1: Idophatic pulmonary fibrosis1458 (Other)
AO2: COPD
AO3: Acute respiratory distress syndrome
AO4: Asthma
ApoptosisPathwayWP254 (WikiPathways)
Carbon monoxide
Chemokine signalling pathwayPathwayWP3929 (WikiPathways)
Cigarette smoke
Emission from biomass fuel and combusiton
Industrial smoke
KE1: Increased oxidative stress1392 (Other)
KE2: Inflammation and pro-fibrotic state 1497 (Other)
KE3: Cell death and cycle arrest
KE4: Mitrochondrial dysfucntion177 (Other)
KE5: Collagen production and reduced ECM
KE6: ER stress
MIE1: ROS production upregulation
Nitrogen oxides
Ozone
Particulate matter
Pesticides
Pollen grains
Profibrotic pathwayPathwayWP3624 (WikiPathways)
ROS damage pathwayPathwayWP5477 (WikiPathways)
Release of pro-inflammatory cytokinesPathwayWP530 (WikiPathways)
SenescencePathwayWP2902 (WikiPathways)
Sulphur oxides

Annotated Interactions

No annotated interactions

Retrieved from "https://classic.wikipathways.org/index.php/Pathway:WP5482"
Personal tools