Oxidative stress in pulmonary tissue (Homo sapiens)

From WikiPathways

Revision as of 15:11, 4 June 2024 by Burritaco (Talk | contribs)
(diff) ←Older revision | Current revision (diff) | Newer revision→ (diff)
Jump to: navigation, search
1Cigarette smokeApoptosisKE3: Cell death and cycle arrestKE2: Inflammation and pro-fibrotic stateAO1: Idophatic pulmonary fibrosisKE1: Increased oxidative stressParticulate matterKE6: ER stressEmission from biomassfuel and combusitonNitrogen oxidesKE4: Mitrochondrial dysfucntionPollen grainsRelease of pro-inflammatory cytokinesAO2: COPDAO3: Acute respiratory distresssyndromeKE5: Collagen production and reduced ECMAO4: AsthmaProfibrotic pathwayOzoneMIE1: ROS production upregulationIndustrial smokeSenescenceROS damage pathwaySulphur oxidesCarbon monoxidePesticidesChemokine signalling pathway


Description

Adverse Outcome Pathway (AOP) for stressor induced increase in ROS production in pulmonary tissue.

Try the New WikiPathways

View approved pathways at the new wikipathways.org.

Quality Tags

Ontology Terms

 

Bibliography

  1. Sajal Chakraborti, Narasimham L. Parinandi, Rita Ghosh, Nirmal K. Gangul, Tapati Chakraborti; ''Oxidative Stress Mechanisms in the Pathogenesis of Environmental Lung Diseases''; Pubmed, 2019 PubMed Europe PMC Scholia

History

CompareRevisionActionTimeUserComment
135313view21:29, 16 August 2024KhanspersOntology Term : 'oxidative stress response pathway' added !
133233view07:50, 10 June 2024BurritacoEndogenous ROS production was deleted and lipid membrane oxidation was removed.
131847view15:11, 4 June 2024BurritacoNew pathway

External references

DataNodes

View all...
NameTypeDatabase referenceComment
AO1: Idophatic pulmonary fibrosis1458 (Other)
AO2: COPD
AO3: Acute respiratory distress syndrome
AO4: Asthma
ApoptosisPathwayWP254 (WikiPathways)
Carbon monoxide
Chemokine signalling pathwayPathwayWP3929 (WikiPathways)
Cigarette smoke
Emission from biomass fuel and combusiton
Industrial smoke
KE1: Increased oxidative stress1392 (Other)
KE2: Inflammation and pro-fibrotic state 1497 (Other)
KE3: Cell death and cycle arrest
KE4: Mitrochondrial dysfucntion177 (Other)
KE5: Collagen production and reduced ECM
KE6: ER stress
MIE1: ROS production upregulation
Nitrogen oxides
Ozone
Particulate matter
Pesticides
Pollen grains
Profibrotic pathwayPathwayWP3624 (WikiPathways)
ROS damage pathwayPathwayWP5477 (WikiPathways)
Release of pro-inflammatory cytokinesPathwayWP530 (WikiPathways)
SenescencePathwayWP2902 (WikiPathways)
Sulphur oxides

Annotated Interactions

No annotated interactions

Retrieved from "https://classic.wikipathways.org/index.php/Pathway:WP5482"
Personal tools