Substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis - Carbon Nanotubes (Homo sapiens)

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Revision as of 21:11, 22 May 2024 by Eweitz (Talk | contribs)
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Accumulation, CollagenIncreased, recruitment of inflammatory cells Loss of alveolar capillary membrane integritySubstance interaction with lung cells Increased, fibroblast proliferation and myofibroblast differentiationTNF-alphaType II Interferon Increased, secretion of proinflammatory mediatorsPulmonary fibrosis Increased, activation of T (T) helper (h) type 2 cellsInflammatory cytokinesApoptosis


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History

CompareRevisionActionTimeUserComment
129804view21:11, 22 May 2024EweitzOntology Term : 'pulmonary fibrosis' added !
129282view12:56, 19 March 2024SamBruss
129281view00:09, 19 March 2024SamBrussNew pathway

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DataNodes

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NameTypeDatabase referenceComment
Increased, activation of T (T) helper (h) type 2 cells
Increased, fibroblast proliferation and myofibroblast differentiation
Loss of alveolar capillary membrane integrity
Accumulation, Collagen
ApoptosisPathwayWP254 (WikiPathways)
Increased, recruitment of inflammatory cells
Increased, secretion of proinflammatory mediators
Inflammatory cytokinesPathwayWP5095 (WikiPathways)
Pulmonary fibrosis
Substance interaction with lung cells
TNF-alphaPathwayWP231 (WikiPathways)
Type II Interferon PathwayWP619 (WikiPathways)

Annotated Interactions

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