Substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis - Carbon Nanotubes (Homo sapiens)

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Pulmonary fibrosisTNF-alphaType II Interferon Increased, recruitment of inflammatory cells Increased, fibroblast proliferation and myofibroblast differentiation Increased, activation of T (T) helper (h) type 2 cellsIncreased, secretion of proinflammatory mediatorsSubstance interaction with lung cells Loss of alveolar capillary membrane integrityAccumulation, CollagenInflammatory cytokinesCollagen biosynthesisApoptosis


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CompareRevisionActionTimeUserComment
129282view12:56, 19 March 2024SamBruss
129281view00:09, 19 March 2024SamBrussNew pathway

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NameTypeDatabase referenceComment
Increased, activation of T (T) helper (h) type 2 cells
Increased, fibroblast proliferation and myofibroblast differentiation
Loss of alveolar capillary membrane integrity
Accumulation, Collagen
ApoptosisPathwayWP254 (WikiPathways)
Collagen biosynthesisPathwayWP2725 (WikiPathways)
Increased, recruitment of inflammatory cells
Increased, secretion of proinflammatory mediators
Inflammatory cytokinesPathwayWP5095 (WikiPathways)
Pulmonary fibrosis
Substance interaction with lung cells
TNF-alphaPathwayWP231 (WikiPathways)
Type II Interferon PathwayWP619 (WikiPathways)

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