Portal:Lipids/PathwayList
From WikiPathways
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Current revision (14:39, 1 October 2020) (view source) |
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| - | === | + | === Existing pathways === |
| - | + | Pathways which have been approved for data analysis can be found on the [https://www.wikipathways.org/index.php/Portal:Lipids/FeaturedPathways Featured pathway] page. | |
| + | We have split these up in 7 groups (Sphingolipids, Eicosanoids, Glycerolipids and Glycerophospholipids, Phosphatidylglycerides, Fatty Acids, Cholesterol, General Pathways related to Lipids). | ||
| - | + | ==== Extend existing (approved) pathways with new literature ==== | |
| - | + | ||
| - | ==== Extend existing pathways with new literature ==== | + | |
* [https://doi.org/10.1002/cm.21067 More proteins], [https://doi.org/10.1002/bies.201300064 localisation], details on regulation of (endocytic) membrane traffic [https://doi.org/10.1111/febs.12116] and [https://doi.org/10.1242/jcs.02855] by phosphoinositide 3-kinases, and [https://doi.org/10.1016/j.febslet.2012.04.046 activity and tumor suppression] thereof for [https://www.wikipathways.org/index.php/Pathway:WP4971 WP4971]. | * [https://doi.org/10.1002/cm.21067 More proteins], [https://doi.org/10.1002/bies.201300064 localisation], details on regulation of (endocytic) membrane traffic [https://doi.org/10.1111/febs.12116] and [https://doi.org/10.1242/jcs.02855] by phosphoinositide 3-kinases, and [https://doi.org/10.1016/j.febslet.2012.04.046 activity and tumor suppression] thereof for [https://www.wikipathways.org/index.php/Pathway:WP4971 WP4971]. | ||
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| + | ==== Update existing (not yet approved) pathways ==== | ||
| + | {|style="background-color:#f4f9fa" | ||
| + | |[[:Pathway:WP4690|Sphingolipid Metabolism (integrated pathway) (mouse)]] | ||
| + | |- | ||
| + | |[[:Pathway:WP4491|Elongation of (very) long chain fatty acids (mouse)]] | ||
| + | |} | ||
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| + | === Create new pathways === | ||
| + | * Public resources for pathway information: [http://lipidmaps.org/ LIPID MAPS] and [http://www.swisslipids.org/ SwissLipids]. | ||
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| + | * See our Help section for [[:Help:New_Contributor_Quickstart|New Contributor QuickStart]], [[:Help:Guidelines_EditorPalette|Editor's Palette Guide]] and [[:Help:Guidelines|Guidelines for Contributors]]. | ||
==== Start a new pathway! ==== | ==== Start a new pathway! ==== | ||
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* [https://doi.org/10.3390/ijms19123931 Class I PI3-K Isoforms in Endometrial Cancer] | * [https://doi.org/10.3390/ijms19123931 Class I PI3-K Isoforms in Endometrial Cancer] | ||
* [https://en.wikipedia.org/wiki/Inositol_trisphosphate_receptor Inositol trisphosphate receptor] | * [https://en.wikipedia.org/wiki/Inositol_trisphosphate_receptor Inositol trisphosphate receptor] | ||
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=== Contact us === | === Contact us === | ||
Current revision
Contents |
Existing pathways
Pathways which have been approved for data analysis can be found on the Featured pathway page. We have split these up in 7 groups (Sphingolipids, Eicosanoids, Glycerolipids and Glycerophospholipids, Phosphatidylglycerides, Fatty Acids, Cholesterol, General Pathways related to Lipids).
Extend existing (approved) pathways with new literature
- More proteins, localisation, details on regulation of (endocytic) membrane traffic [1] and [2] by phosphoinositide 3-kinases, and activity and tumor suppression thereof for WP4971.
Update existing (not yet approved) pathways
| Sphingolipid Metabolism (integrated pathway) (mouse) |
| Elongation of (very) long chain fatty acids (mouse) |
Create new pathways
- Public resources for pathway information: LIPID MAPS and SwissLipids.
- See our Help section for New Contributor QuickStart, Editor's Palette Guide and Guidelines for Contributors.
Start a new pathway!
- Phosphoinositide Signaling
- PI3-K in Human diseases
- Class I PI3-K Isoforms in Endometrial Cancer
- Inositol trisphosphate receptor
Contact us
Send questions about WikiPathways to the wikipathways-discuss mailing list.
How to add a pathway to the portal
The list of Featured Pathways is not static and can be updated at any time. If you know of a pathway that should be added, please contact the administrator.
